I was talking with Dennis Burg and told him about a resume we should all have ready  to show our doctors at the VA and our civilian doctors.  Since our toxin legacy is a small portion of a once in generation exposure.



Toxin Resume for our I Corps Veterans


I was exposed to dioxin specifically (TCDD) by mouth, lung, and skin exposures for at least one year.  TCDD acts like a hormone and can modify: cell nuclei and DNA, enzymes, other hormones, protein, metabolism, and ones blood makeup, all at microbiological level.  At system level it is noted for medical issues in the following:  some cancers, heart disease, vascular disease, neurological ailments, endocrine disturbances, and hematological difficulties


I was exposed to a second component at the same time as the TCDD in the form of 2,4,-D.  This 2,4-D according to the EPA can cause:  central nervous system damage and liver damage.


I was exposed to a second chemical composition within the same time frame for over a year as the above TCDD exposure of 2,4-D and Picloram.  Picloram also contained two other toxins Hexachlorobenzene and Nitrosamine.


These two, Hexachlorobenzene and Nitrosamine are noted for the following:  mortality from cancers of the esophagus, oral cavity, and pharynx.  When used in pesticides or herbicides Nitrosamine may cause DNA damage and cell death.  Hexachlorobenzene is also noted for effects on the liver, nervous system, and immune function at low doses.


I was exposed to a third toxin within the same time frame for over a year called cacodylic acid.  This toxin is a noted neurotoxin and can cause the following:


Garlic odor of breath and feces, metallic taste in mouth.

Adverse GI effects predominate with vomiting, abdominal pain and rice-water or bloody diarrhea. 

GI effects may also include inflammation, vesicle formation and eventual sloughing of the mucosa in the mouth, pharynx and esophagus. 

Central nervous system effects are that are common include headache, dizziness, drowsiness and confusion.

Symptoms may progress to include muscle weakness and spasms, hypothermia, lethargy, delirium, coma and convulsions.

Renal injury manifests as proteinuria, hematuria, glycosuria, oliguria, and shows in the urine. In severe poisoning cases, acute tubular necrosis results.

Cardiovascular effects include shock, cyanosis and cardiac arrhythmia.

Elevated liver enzymes and jaundice may manifest producing liver damage.

Injury to blood-forming tissues may cause anemia, leucopenia and thrombocytopenia.

Muscle weakness, fatigue, anorexia, weight loss.

Hyperpigmentation, hyperkeratosis.

Peripheral neuropathy, paresthesia, paresis and ataxia.

Inability to coordinate voluntary muscular movements.

Subcutaneous edema in face, eyelids, and ankles.

Stomatitis, white striations across the nails (Mees lines) and sometimes loss of nails or hair.

Liver toxicity as indicated by hepatomegaly, jaundice, and cirrhosis.

Renal toxicity leading to oliguria, proteinuria, and hematuria.

EKG abnormalities and peripheral vascular disease.

Hematologic abnormalities.



At the same time I was exposed to all of these chemicals and chemical compositions, I was taking a pill called Dapsone.  This is a leprosy treatment pill.  The side effects of this pill are:


Blood Effects

Dose-related hemolysis is the most common adverse effect and is seen in patients with or without G6PD deficiency.  {Glucose 6-phosphate dehydrogenase (G6PD) deficiency is an enzyme deficiency of the red blood cells.  G6PD deficiency leads to an abnormal rupture (breakage) of the red blood cells called hemolytic anemia (abnormally low red blood cell count).}  Almost all patients demonstrate the inter-related changes of a loss of 1-2 g of hemoglobin, an increase in the reticulocytes (2-12%), a shortened red cell life span and a rise in methemoglobin.  G6PD deficient patients have greater responses.


Nervous System Effects

Peripheral neuropathy is a definite but unusual complication of Dapsone therapy in non-leprosy patients.  Motor loss is predominant.  If muscle weakness appears, Dapsone should be withdrawn.  Recovery on withdrawal is “usually substantially complete”.  The mechanism of recovery is reported by axonal regeneration.  Some recovered patients have tolerated re treatment at reduced dosage.


In addition to the warnings and adverse effects reported above, additional adverse reactions include: nausea, vomiting, abdominal pains, pancreatitis, vertigo, blurred vision, tinnitus, insomnia, fever, headache, psychosis, photo toxicity, pulmonary eosinophilia, tachycardia, albuminuria, the nephrotic syndrome, hypoalbuminemia without proteinuria, renal papillary necrosis, male infertility, drug-induced Lupus erythematosus and an infectious mononucleosis-like syndrome. 


In addition to the other herbicides and mixtures of herbicides I was also exposed to great quantities of Malathion (S- [1,2-dicarbethoxyethyl]-0, 0-dimethyldithiophosphate).


Could you please tell me what is wrong with me and which one of the above toxins or combinations of toxins possibly caused whatever it is that is wrong with me? 


In addition, could you please rule out any synergy effects that may have happened between the chemicals that have never been studied as to their never discovered medical outcomes and/or confirmed?


That about sums it up for the Vietnam Veteran in I Corps.


I think the average person can now see that studies directed at any one specific toxin to any form of a dose response to satisfy the VA and the NAS/IOM for compensations is totally useless and just a government stalling tool to limit Veterans compensations and expenditures.


Remember fellows, the VA only gets away with what the president and congress allows them to do, which now borders on the criminal for forty years now.


Our politicians and presidents have learned that by “creating the veteran”, they then indeed did and do “put aside the citizen” and any rights that the most “noble of all citizens” has to any truth, fairness, and constitutional justice in our nation.   



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